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Winter2dill

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E the antibody isotype-profile of influenza-specific B cell responses. TLR7 plus type I IFN signaling can drive class switch recombination to IgG2a/c, while its stimulation without type I IFNR signaling drives IgG1 (75). This is consistent with studies showing that a lack of type I IFN direct signaling following influenza infection decreases IgG2a/c and increases IgG1 (30). Recently, TLR7 signalin
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G chlorophyll.Fig. 3. A schematic of the putative carotenoid biosynthesis pathway in Acaryochloris. The reaction catalyzed by CrtR passes through the intermediate -cryptoxanthin.which likely provided the origin for CAO, and other poorly understood enzymes, such as the phylogenetically aberrant divinyl chlorophyllide reductase (33), which shows an unusual phylogenetic topology in A. marina (AM1 239
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G chlorophyll.Fig. 3. A schematic of the putative carotenoid biosynthesis pathway in Acaryochloris. The reaction catalyzed by CrtR passes through the intermediate -cryptoxanthin.which likely provided the origin for CAO, and other poorly understood enzymes, such as the phylogenetically aberrant divinyl chlorophyllide reductase (33), which shows an unusual phylogenetic topology in A. marina (AM1 239
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Ecies (32). Such enzymes could provide another means for Chl d biosynthesis. A. marina codes for 12 proteins with putative radical SAM motifs, far more than expected from an oxygen-producing cyanobacterium. Of these 12, two (AM1 5023 and AM1 5798) share very little homology with other sequenced cyanobacteria. The Chl d biosynthesis pathway may not be as simple as expected. Unlike the formyl side c
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Ecies (32). Such enzymes could provide another means for Chl d biosynthesis. A. marina codes for 12 proteins with putative radical SAM motifs, far more than expected from an oxygen-producing cyanobacterium. Of these 12, two (AM1 5023 and AM1 5798) share very little homology with other sequenced cyanobacteria. The Chl d biosynthesis pathway may not be as simple as expected. Unlike the formyl side c
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Is that between pREB7 and pREB8, with 29 of pREB7 (or 38 of pREB8) nucleotides sharing 75 identity. The matching regions range from 100 to 8,500 bp with an average of 1,800 bp. Several plasmids also share a few very large homologous regions ( 10 kbp) but do not share a significant global sequence identity. The internal homology between all A. marina plasmids exceeds that seen between its plasmi
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Is that between pREB7 and pREB8, with 29 of pREB7 (or 38 of pREB8) nucleotides sharing 75 identity. The matching regions range from 100 to 8,500 bp with an average of 1,800 bp. Several plasmids also share a few very large homologous regions ( 10 kbp) but do not share a significant global sequence identity. The internal homology between all A. marina plasmids exceeds that seen between its plasmi
1
E the antibody isotype-profile of influenza-specific B cell responses. TLR7 plus type I IFN signaling can drive class switch recombination to IgG2a/c, while its stimulation without type I IFNR signaling drives IgG1 (75). This is consistent with studies showing that a lack of type I IFN direct signaling following influenza infection decreases IgG2a/c and increases IgG1 (30). Recently, TLR7 signalin