Herefore be expected to overexpress rsbN, the resulting increase in anti-BldN activity might interfere with the expression of BldN-dependent genes and contribute significantly to the bald phenotype of bldD mutants. The most well-studied target of BldN is bldM, which encodes an orphan response regulator (Molle Buttner,FEMS Microbiol Rev 38 (2014) 345??2013 The Author. FEMS Microbiology Reviews pu